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Moreover, administration of human islet amyloid polypeptide in diabetic mice was found to induce -cell dysfunction, concomitant with activation of TRPV4 channels and subsequent ERS induction (Liu et al., 2022)

References 47

10.1007/BF03033334

FMS-like Receptor Tyrosine Kinase 3 internal tandem duplication (FLT3-ITD) mutations create ferroptosis sensitivity through altered polyunsaturated fatty acid metabolism [15], while nucleophosmin 1 (NPM1) mutated AML exhibits exceptional cystine dependency [14]